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Ich habe ja auch nicht gesagt man solle sich hauptsächlich von Schokolade ernähren, es war eben die Sache, dass hier im Forum Schokolade oft als Hauptgrund genannt wurde warum LC bei manchen nicht funktioniert, bei mäßigem Schokoladenkonsum halte ich dies allerdings für ausgeschlossen. Da halte ich andere Lebensmittel, die bei LClern beliebt sind, für problematischer, Wurst z.B.. Nein ich bin doch nicht aufgebracht, ich wollte nur die gute Schoki verteidigen. Wo wir schon bei Schokolade sind können wir auch gleich Kaffee ansprechen, hier scheiden sich ja die Geister, immer wieder wird er als schlecht diffamiert, aber Ray Peat hingegen hält Kaffee wieder für völlig unproblematisch. Vlt. weiß ja jemand mehr dazu. |
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http://inhumanexperiment.blogspot.co...e-depends.html
When it comes to storing dietary fat into fat tissue, are all fatty acids created equal? Recently, I've been trying to find out more information on how different types of fats are absorbed and stored as adipose tissue. In particular, I would like to see direct comparisons between saturated, monounsaturated and polyunsaturated fat and how they affect weight and fat gain in humans when energy intake is kept constant. So far, I haven't had much luck finding such studies. What I did find is an old paper that looked into how different dietary fatty acids relate to fatty acids in adipose tissue (link). In other words, the authors studied whether the ratio of saturated and unsaturated fats in fat tissue is similar to the ratio of fats obtained from diet. To answer this question, they first starved rabbits (rabbit starvation, anyone?) for 3-4 weeks to deplete their fat stores and then put them on diets with varying fatty acids to see how it affected fatty acid distributions in adipose tissue. Fatty acid composition of the diets Five different diets were used, each one containing 20% by weight of either palm oil, cocoa butter, safflower oil, linseed oil (also known as flax oil) or rapeseed oil. These fats were chosen because each one is rich in a particular fatty acid: palm oil in palmitic acid, cocoa butter in stearic acid, safflower oil in linoleic acid, linseed oil in linolenic acid, and rapeseed oil in erucic acid. In addition, a low-fat control diet containing only 2.2% fat was used. All rabbits were fed ad libitum. Though rabbits presumably consume little fat in the wild, there appear to be no special problems with feeding fat to rabbits (link, link). Rabbits eating a diet high in fat do gain more weight, however. The figure above shows the percentages of saturated (SA), monounsaturated (MUFA) and polyunsaturated (PUFA) fatty acids in each diet. As you can see, palm oil and cocoa butter are mostly saturated fat, safflower and linseed oil are mostly polyunsaturated fat, and rapeseed oil is mostly monounsaturated fat. Saturated and unsaturated fatty acids in adipose tissue When you feed animals various kinds of fat, you expect to see those same kinds of fat being deposited into their fat tissue, right? Saturated fat being stored as saturated fat, monounsaturated as monounsaturated and polyunsaturated as polyunsaturated. That seems logical enough but is apparently not the case. The rabbits on diets high in saturated fat resisted the deposition of large amounts of saturated fatty acids in their fat tissue. In fact, the rabbits eating diets high in palm oil and cocoa butter had similar levels of saturated fatty acids in their adipose tissue as rabbits eating the low-fat control diet. This despite the fact that the palm oil and cocoa butter diets not only contained much more fat as a whole, but also more than twice the percentage of saturated fat relative to total fat intake than in the control diet. The graph above shows the percentages of fatty acids in adipose tissue after each diet. As you can see, feeding the rabbits palm oil, cocoa butter or rapeseed oil resulted in a distribution that is similar to the result seen from eating the control diet. Feeding them safflower oil or linseed oil, on the other hand, resulted in a significantly different distribution, one that is high in PUFAs but low in MUFAs and SAs. The plateau of dietary saturated fat deposition into adipose tissue does not seem to be a temporary phenomenon. To find out how long-term administration of saturated fat affected fat deposition, the authors fed two additional rabbits a diet high in cocoa butter for a year. These rabbits showed similar levels of saturated fatty acids in their fat tissue as the rabbits that consumed the same diet for only a month. In other words, saturated fat in their adipose tissue did not increase further even with prolonged intake of cocoa butter. Individual fatty acids in adipose tissue So the distribution of unsaturated and saturated fats in adipose tissue depends on the fat composition of the diet. As we've seen, the relationship is stronger for polyunsaturates than it is for monounsaturates and especially saturates. What about individual fatty acids? The same discrepancy between dietary intake and adipose tissue deposition is apparent here. The palm oil diet was 164% higher in palmitic acid than the control diet, but the concentration of palmitic acid in adipose tissue was only 30% higher in the palm oil group. Similarly, the cocoa butter diet contained eight times as much stearic acid as the control diet, but stearic acid levels in the cocoa butter group were only 52% higher. Again, things were a little different in the polyunsaturated department. The percentage of linoleic acid in the adipose tissue more than doubled on the safflower oil diet compared to the control diet, and the linolenic acid concentration in adipose tissue was 31.1% on the linseed oil diet compared to 3.9% on the control diet. It seems that out of the six individual fatty acids studied, oleic acid (primarily found in cocoa butter and palm oil) and linoleic acid (primarily found in safflower oil) were deposited most effectively. Oleic acid (a monounsaturated fat) in adipose tissue constantly exceeded the amount of oleic acid present in the diets. Erucic acid (mainly in rapeseed oil) and stearic acid (mainly in cocoa butter) showed the least deposition. Conclusion and discussion Adipose tissue takes up many different fatty acids from the diet. The degree to which these fatty acids are absorbed varies, however. Specifically, a diet high in saturated fat raises saturated fat in rabbit adipose tissue only modestly, whereas a diet high in polyunsaturates increases PUFA levels in adipose tissue dramatically. Several reasons may explain these differences in dietary and adipose tissue fat composition. First, the authors note that since the control rabbits did have saturated fat in their adipose tissue despite consuming a diet very low in fat, they presumably synthesized saturated fat from carbohydrates in the diet. Therefore, when the intake of certain fatty acids is low, the body adapts by producing them through other means, using carbohydrates or other fatty acids. Second, when the intake of certain fatty acids exceeds certain limits, the body is able to convert dietary fats from one form into another. According to the authors, retroconversion (chain-shortening) and desaturation of dietary fats may be a necessary function of the body to ensure optimum cell membrane fluidity. That is, when saturated fat intake is very high, the body converts some of the saturated fat into unsaturated fat to keep things working properly. This mechanism does not appear to be in place in rabbits for polyunsaturated fats, however. The fatty acid ratios in adipose tissue appear to reflect those in the diet very strongly when the diet is high in PUFAs. This makes me wonder whether a similar effect happens in other mammals, especially humans. The authors do not report whether calorie intake varied between groups or how much actual adipose tissue the groups had. They only mention that during the 4-week experiment, all rabbits in the five experimental groups were healthy and gained weight. Geändert von pm2 (13.07.2009 um 14:52 Uhr) |
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Ab und zu mische mir sehr gerne das entölte und zuckerfreie
Demeter Bio-Kakaopulver vom Hersteller Rapunzel in Bio-Milch, Sahne ggf. auch Butter. Dient für mich als Ersatz für Süssigkeiten und schmeckt auch ohne Zucker richtig gut. Ich denke Kakao in Maßen und nicht in Massen ist durchaus positiv für den Menschen, es sei denn man verträgt es nicht. |
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Zitat:
Hier übrigens eine Studie, die Glucose und Fruktose vergleicht: Consuming fructose-sweetened, not glucose-sweetened, beverages increases visceral adiposity and lipids and decreases insulin sensitivity in overweight/obese humans Natürlich mit entsprechend schlechtem Abschneiden von Fruktose. |
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Zitat:
Interessante Studie nicht wahr. Ich hatte diese bereits schon auf Seite 542 (Reine Haut durch kohlenhydratarme Ernährung) verlinkt Damals auch schon mit unrühmlichen Ausgang für Fruktose.@pm2 Sehr interessante Studie, wenn auch leider an Ratten durchgeführt. Muss ich mir bei Gelegenheit mal genauer anschauen. Danke. |
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Wie schädlich sind Nudeln eigentlich?
Ich frage, weil hier in der WG essen sie alle Nudeln (fast jeden Tag) und haben die reinste Haut die es auf Erden gibt!
__________________
! Akne geheilt ! durch Lösung aller Ursachen. Die Ursachen für Akne: - Kalium-Mangel - Demodex-Milben in der Haut - schlechtes Darmmillieu (Verschlackung, wenig gute Darmbakterien..) - Lebensmittelunverträglichkeiten (hauptsächlich Milchprodukte) - Toxische Stoffe (Schwermetalle, Pilze im Körper wie z.B. Candida, Schweinefleisch) - Rasur-Pickel bedingt durch unzureichende Desinfektion Keine Schulmedizin-Scharlatanerie! Es ist SO einfach! |
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Ich ess jetzt auch jeden tag Nudeln,und mir machen die auch nix aus.Im gegenteil,meine verdauung ist viel besser,und ich kann jeden tag aufs Klo.Reis hat bei mir sehr gestopft,das ich manchmal soagr 3 tage nicht auf die Toilette konnte.Ausserdem schmecken mir Nudeln auch viel besser.
Ps.Barilla Forever*g*. |
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